Sirope de maíz de alta fructosa
GI(Glucose = 100)
Estudio único: un solo estudio publicado, no una media agrupada.
Principal · 56 GI
Wright, Ellis & Ilag — HFCS GI control in filtered-molasses study (Plant Foods Hum Nutr) (2014)
Energy(kcal/100g)
Principal · 281 kcal/100g
USDA FoodData Central SR Legacy — Syrups, corn, high-fructose (NDB 19351 / FDC 169659) (2018)
Carbs(g/100g)
Principal · 76 g/100g
USDA FoodData Central SR Legacy — Syrups, corn, high-fructose (NDB 19351 / FDC 169659) (2018)
Sugars(g/100g)
Principal · 75.6 g/100g
USDA FoodData Central SR Legacy — Syrups, corn, high-fructose (NDB 19351 / FDC 169659) (2018)
Beneficios funcionales
Todavía no hay minerales citados ni otras notas funcionales.
Los minerales citados pueden sumar hasta +2 en El mejor endulzante para el día a día — ver Methods.
Riesgos y efectos secundarios
Contenido elevado de fructosa
caution
Hepatic / free-sugars framing (WHO). Low GI is not a hepatic free pass. Composition and use-level matter. Not a treatment claim. Scored through the fructose term on every list; above 50% free fructose, every list caps the score at 30, and Weight goals keeps it at or below table sugar (Diabetic-friendly leaves these rows out).
WHO — Guideline: sugars intake for adults and children (2015)
Visceral fat gain vs glucose (10-week trial)
caution
J Clin Invest 2009 randomized trial, 32 overweight or obese adults, 25% of energy as fructose or glucose drinks for 10 weeks: same weight gain, but visceral fat rose only on fructose. Compares fructose with glucose, not with table sugar: sucrose drinks also raised visceral fat over 6 months (Am J Clin Nutr 2012), and swapping fructose for other carbohydrate at equal calories did not change weight (Ann Intern Med 2012 meta-analysis). Scored: on Weight goals a row above 50% free fructose stays at or below table sugar and ranks after it — our editorial reading, since it replaces sucrose's glucose half with fructose.
Stanhope et al. — Consuming fructose-sweetened, not glucose-sweetened, beverages increases visceral adiposity and lipids and decreases insulin sensitivity in overweight/obese humans (J Clin Invest) (2009)
Formación acelerada de productos finales de glicación avanzada (AGE, in vitro)
caution
Fructose often ~7.5–10× more reactive than glucose in non-enzymatic glycation assays of model proteins — not “10× AGEs in blood.” Plasma fructose is normally far lower than glucose; in-vivo vascular AGE evidence is incomplete (Schalkwijk). Not scored separately — the fructose term already covers high-fructose rows.
Aragno & Mastrocola — Dietary sugars and endogenous AGE formation (Nutrients) (2017)
No es consejo médico. Las penalizaciones publicadas, cuando existen, se explican en Cómo puntuamos y no representan una escala clínica de gravedad.
Notas
Low GI measures blood-glucose rise after a load — not liver fructose handling or glycation chemistry. Pure fructose and high-fructose syrups can still sit under free-sugars / hepatic-load framing, and in-vitro fructation runs faster than glucation, even when the GI cell looks green.
Editorial table note. Not a diagnosis or treatment claim. Everyday recipe applies a published high-fructose penalty (−0.30).
WHO — Guideline: sugars intake for adults and children (2015)
Wright 2014 measured GI 56 for an HFCS control. Brand-Miller (GI News 2010) noted manufacturers had not published graded HFCS-42 vs HFCS-55 assays; blogs quoting 58–73 by grade are estimates, not this cell.
Composition (42 vs 55) still shifts expected GI.
GI News / Brand-Miller — HFCS GI not published by manufacturers (University of Sydney) (2010)
Las notas explican lo que el eje abierto o las etiquetas de riesgo por sí solos no muestran; no es consejo médico.
Una celda vacía indica que aún no disponemos de una fuente que describa el método. El primer puesto responde a la regla publicada, no a un veredicto médico.